The Pathogenesis of Memory and Its Causes

Abstract

Arnaldo PG de Paiva Neto

Objective: to evaluate the memory phenomenon when synchronizing an area of the brain interacting with the external environment. Introduction: Dr. Dale Bredesen estimates that there will be more than 160 million individuals with Alzheimer’s dementia in the world by 2050. Amnesia in temporal lobe epilepsy indicates that the hyperactive excitability generated in this temporal lobe impairs the ability to memorize, destabilizing the rhythm in relative to the other brain lobes.

Methodology: through literature review it is assessed that there is a working relationship between the region on the cortical side of the brain and the contralateral homotopic cortex.

Results and discussion: the types of amnesia are classified in a first group whose organic causes predominate. We must not forget the danger of the accumulation of non-functional proteins that can precede the formation of beta-amyloid aggregates. They replicate more intensely than an infectious agent does, because they do not need genetic material for their multiplication.

Conclusion: the first group may also be due to the lack of memory consolidation (sleep disorders, lack of mnemic exercise, malnutrition, infection or other conditions). There is a second group whose psychological causes predominate. Studies on the hormone DHEA (dehydroepiandrosterone) can help improve these neurodegenerative processes.

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