Tuberculous arachnoiditis is a generally regular reason for myeloradiculopathy in nations endemic for tuberculosis. The provocative exudate encompasses, however doesn't penetrate, the spinal rope and nerve roots. Much of the time, there is vascular inclusion with peri-arteritis and impediment of little vessels. Neuronal structures are harmed by direct pressure just as by ischaemia. The progressions of arachnoiditis might be central, multifocal, or diffuse. In tuberculous arachnoiditis highlights of spinal line or nerve root inclusion may prevail however frequently there is a blended picture. Often, there is clinical proof of multifocal radiculo myelopathy, yet in any event, when meningeal inclusion is broad, manifestations may emerge from a solitary level. The sign of conclusion is the trademark myelographic picture, demonstrating poor progression of balance material with different unpredictable filling surrenders, growth development, and now and again spinal square. Once in a while, myelography might be typical. The CSF changes are those of interminable meningitis, much of the time CSF sugar fixation is ordinary. Once in a while lumbar tap might be dry. These patients need sufficient enemy of tuberculous treatment for at any rate one year. The job of corticosteroids is questionable, however there are a few reports of clearly checked improvement following corticosteroid organization. In the event that the patient doesn't react to clinical treatment, medical procedure might be required.