Numerous lines of studies have exhibited the postprandial hypertriglyceridemia as one of the hazard factors for cardiovascular maladies in stout people [3]. Anyway it has been muddled why hefty patients with polycystic ovarian condition (PCOS) have higher occurrence of cardiovascular illnesses. T. K. Tun et al. have a thoughtfulness regarding, specifically, the settings of the postprandial changes in the lipid profile by examination of two large gatherings of PCOS or non-PCOS. Lipids assume significant jobs in the body to store vitality and as the parts of organic layers, steroid hormones, bile acids, nutrients, etc. They are provided from abstains from food or from the once more combination in the liver. Unsaturated fats fundamentally put away as triglycerides are the significant vitality hotspot for muscle and heart. Be that as it may, the overproduction and gathering of triglycerides in fat tissue and different tissues are firmly identified with the human metabolic issue. Unsettling influence of cholesterol homeostasis is additionally firmly identified with atherosclerosis. Skeletal muscle is one of the primary controllers of starch and lipid digestion in our living being, and subsequently, it is profoundly vulnerable to changes in glucose and unsaturated fat (FA) accessibility. Skeletal muscle is a very unpredictable tissue: its metabolic limit relies upon the kind of filaments it is comprised of and the degree of incitement it experiences, for example, intense or ceaseless withdrawal.